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"The chemical mechanism of oxidative stress due to the non-transferrin-bound iron (NTBI)"
written by Anatole A. Klyosov, Igor L. Rozhanskii,
published by Advances in Bioscience and Biotechnology, Vol.3 No.7, 2012
has been cited by the following article(s)：
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[1] New Insight into Toxicity due to Oligomeric Amyloid-b Peptide and Alpha-Synuclein Protein induced by Copper (II) ion
International Journal of Chemistry 2014[2] Model Reactions for the Formation of Iron Deposition
International Journal of Chemistry 2014[3] 鉄化合物の構造予測と発ガン性
Journal of Computer Chemistry, Japan 2015[4] Abnormal iron accumulation in brain and new iron chelator for labile iron removal therapy
International Journal of Chemistry 2015[5] IRON CHELATORS FOR LABILE IRON REMOVAL THERAPY AND ENHANCEMENT OF LONGEVITY
European Journal of Biology and Medical Science Research 2015[6] Investigated and available therapeutic options for treating aceruloplasminemia
Expert Opinion on Orphan Drugs 2015[8] New Insight into Toxicity due to Oligomeric Amyloid-[Beta] Peptide and Alpha-Synuclein Protein induced by Copper (II) ion
International Journal of Chemistry 2015[9] Abnormal Iron Accumulation in Brain and New Iron Chelator for Labile Iron Removal Theraphy
International Journal of Chemistry 2015[10] 鉄代謝異常の解明に向けて~ 新たな創薬のターゲット~
神経治療学 2016[11] Current insights on the role of iron and copper dyshomeostasis in the pathogenesis of bilirubin neurotoxicity
Life Sciences 2017[12] A novel, nontoxic iron chelator, super-polyphenol, effectively induces apoptosis in human cancer cell lines
Oncotarget 2018[13] Iron-mediated oxidative cell death is a potential contributor to neuronal dysfunction induced by neonatal hemolytic hyperbilirubinemia
Archives of Biochemistry and Biophysics 2018 - No relevant information.