I. Marczinovits, L. Kovacs, A. Gyorgy, G. K. Toth, L. Dorgai, J. A. Molnar, “A peptide of human muscarinic acetylcholine receptor 3 is antigenic in primary Sj?gren’s syndrome”. Journal of Autoimmunity, Vol. 24, 2005, pp. 47-54.
 H. M. Moutsopoulos, N. Talal, “Immunologic abnormalities in Sjogren syndrome”. In: Sj?gren’s syndrome: clinical and immunopathological aspects. N. Talal, H. M. Moutsopoulos, editors. Berlín: Springer-Verlag; 1987, pp 258-265.
 C. Perez Leiros, L. Sterin-Borda, E. Borda, J. C. Goin, M. M. Hosey, “Desensitization and sequestration of human M3 muscarinic acetylcholine receptors by autoantibodies from patients with Chagas’ disease”. Journal of Biology and Chemeistry Vol. 272, 1999, pp. 12989-12993.
 S. Bacman, L. Sterin-Borda, J. J. Camusso, R. Arana, O. Hubscher, E. Borda, “Circulating antibodies against rat parotid gland M3 muscarinic receptors in Sj?gren syndrome”. Clinical Immunology, Vol. 104, 1996, pp. 454- 459.
 S. Bacman, A. Berra, L. Sterin-Borda, E. Borda, “Muscarinic acetylcholine receptor antibodies as a new marker of dry eye Sjogren syndrome”. Investigative Ophthalmology Vision Science, Vol. 42, 2001, pp. 321-327.
 S. Reina, L. Sterin-Borda, B. Orman, E. Borda, “Autoantibodies against submandibular gland musdcarinic cholinoceptor subtypes in primary Sjogren syndrome”. European Journal of Inflammation, Vol. 3, 2005, pp. 135-141.
 L. Kovacs, I. Marczinovits, A. Gryorgy, G. Toth, L. Dorgai, J. Pal, “Clinical associations of autoantibodies to human muscarinic acetylcholine receptor M3 in primary Sj?gren syndrome”. Rheumatology, Vol. 44, 2005, pp. 1021-1025.
 S. Reina, L. Sterin-Borda, B. Orman, E. Borda, “Human mAChR antibodfies from Sjogren syndrome sera increase cerebral nitric oxide synthase activity and nitric oxide synthase mRNA level”. Clinical Immunology, Vol. 113, 2004, pp. 193-202.
 L. J. Dawson, J. Stanbury, N. Venn, B. Hasdimir, S. N. Rogers, P. M. Smith, “Antimuscarinic antibodies in primary Sjogren’s syndrome reversibly inhibit the mechanism of fluid secretion by human submandibular salivary acinar cells”. Arthritis Rheumatology, Vol. 54, 2006, pp. 1165-1173.
 J. Li, Y. M. Ha, N. Y. Ku, S. Y. Choi, S. J. Lee, S. B. Oh, et al. “Inhibitory effects of autoantibodies on the muscarinic receptors in Sjogren’s syndrome”. Laboratory Investigation, Vol. 84, 2004, pp.1430-1438.
 S. A. Waterman, T. P. Gordon, M. Rischmueller, “Inhibitory effects of muscarinic receptor autoantibodies on parasympathetic neurotransmission in Sjogren’s syndrome”. Arthritis Rheumatology, Vol. 43, 2000, pp. 1647-1654.
 S. Cha, E. Singson, J. Cornelius, J. P. Yagna, H. J. Knot, A. B. Peck, “Muscarinic acetylcholine type-3 receptor desensitization due to chronic exposure to Sj?gren syndrome-associated autoantibodies”. Journal of Rheumatology, Vol. 33, 2006, pp. 296-306.
 J. Yuan, A. S. Bowman, M. Aljamali, M. R. Payne, J. S. Tucker, J. W. Dillwith, et al. “Prostaglandin E(2)-stimulated secretion of protein in salivary glands of the lone star tick via a phosphoinositide signaling pathway”. Insect Biochemical Molecular Biology, Vol. 30, 2000, pp. 1099-1106.
 G. M. Pasinetti, “Cyclooxygenase and Alzheimer’s disease: implications for preventive initiatives to slow the progression of clinical dementia”, Archives of Gerontology and Geriatric, Vol. 33, 2001, pp. 13-28.
 V. Mollage, C. E. Muscoli, E. Masini, S. Cuzzocrea, D. Salvemini, “Modulation of prostaglandin biosynthesis by nitric oxide and nitric oxide donors”. Pharmacological Review, Vol. 57, 2005, pp. 217-252.
 S. Ganzinelli, E. Borda, L. Sterin-Borda, “Autoantibodies from schizophrenia patients induce cerebral cox-1/iNOS mRNA expression with NO/PGE2/MMP-3 production”. International Journal of Neuropsychopharmacology, Vol. 13, 2010, pp. 293-303.
 B. Nicke, K. Detjen, C. D. Logsdon, “Muscarinic cholinergic receptors activate both inhibitory and stimulatory growth mechanisms in NIH3T3 cells”. Journal of Biology and Chemistry, Vol. 274, 1999, pp. 21701-21706.
 J. S. Gutkind, K. C. Robbins, “Activation of transforming G protein-coupled receptors induces rapid tyrosine phosphorylation of cellular proteins, including p125FAK and the p130 v-src substrate”. Biochemistry and Biophysic Research Communication, Vol. 188, 1992, pp.155-161.
 S. Gupta, T. Barrett, A. J. Whitmarsh, J. Cavanagh, H. K. Sluss, B. Dérijard, R. J. Davis, “Selective interaction of JNK protein kinase isoforms with transcription factors”. EMBO Journal, Vol. 15, 1996, pp. 2760-2770.
 J. M. Kyriakis, P. Banerjee, E. Nikolakaki, T. Dai, E. A. Rubie, M. F. Ahmad, J. Avruch, J. R. Woodgett. “The stress-activated protein kinase subfamily of c-Jun kinases”. Nature, Vol. 369, 1994, pp. 156-160.
 A. Paul, S. Wilson, C. M. Belham, C. J. Robinson, P. H. Scott, G. W. Gould, R. Plevin, “Stress-activated protein kinases: activation, regulation and function”. Cellular Signalling, Vol. 9, 1997, pp. 403-410.
 J. S. Gutkind, “The pathways connecting G protein-coupled receptors to the nucleus through divergent mitogen- activated protein kinase cascades”. Journal of Biology and Chemistry, Vol. 273, 1998, pp. 1839-1842.
 O. A. Coso, M. Chiariello, G. Kalinec, J. M. Kyriakis, J. Woodgett, J. S. Gutkind. “Transforming G protein-coupled receptors potently activate JNK (SAPK). Evidence for a divergence from the tyrosine kinase signaling pathway”. Journal of Biology and Chemistry, Vol. 270, 1995, pp. 5620-5624.
 F. M. Mitchell, M. Russell, G. L. Johnson, “Differential calcium dependence in the activation of c-Jun kinase and mitogen-activated protein kinase by muscarinic acetylcholine receptors in rat 1a cells”. Biochemistry Journal, Vol. 309, 1995, pp. 381-384.
 O. A. Coso, H. Teramoto, W. F. Simonds, J. S. Gutkind, “Signaling from G protein-coupled receptors to c-Jun kinase involves beta gamma subunits of heterotrimeric G proteins acting on a Ras and Rac1-dependent pathway”. Journal of Biology and Chemistry, Vol. 271, 1996, pp. 3963-3696.
 M. T. Ramirez, V. P. Sah, X. L. Zhao, J. J. Hunter, K. R. Chien, J. H. Brown, “The MEKK-JNK pathway is stimulated by alpha1-adrenergic receptor and ras activation and is associated with in vitro and in vivo cardiac hypertrophy”. Journal of Biology and Chemistry, Vol. 272, 1997, pp. 14057-14061.
 P. S. Shapiro, J. N. Evans, R. J. Davis, J. A. Posada, “The seven-transmembrane-spanning receptors for endothelin and thrombin cause proliferation of airway smooth muscle cells and activation of the extracellular regulated kinase and c-Jun NH2-terminal kinase groups of mitogen-activated protein kinases”. Journal of Biology and Chemistry, Vol. 271, 1996, pp. 5750-5754.
 M. A. Bogoyevitch, A. J. Ketterman, P. H. Sugden, “Cellular stresses differentially activate c-Jun N-terminal protein kinases and extracellular signal-regulated protein kinases in cultured ventricular myocytes”. Journal of Biology and Chemistry, Vol. 270, 1995, pp. 29710-29717.
 C. Vitali, S. Bombardieri, H. M. Moutsopoulos, G. Balestrieri, W. Bencivelli, R. M. Bernstein, et al. “Preliminary criteria for the classification of Sj?gren’s syndrome. Results from a prospective concerted action supported by the European Community”. Arthritis Rheumatology, Vol. 36, 1993, pp. 340-347.
 T. Borda, A. Genaro, L. Sterin-Borda, G. Cremaschi, “Involvement of endogenous nitric oxide signalling system in brain muscarinic acetylcholine receptor activation”. Journal of Neural Transmission, Vol. 105, 1995, pp. 193- 204.
 P. G. Wylie, R. A. Challiss, J. L. Blank, “Regulation of extracellular-signal regulated kinase and c-Jun N-terminal kinase by G-protein-linked muscarinic acetylcholine receptors”. Biochemistry Journal, Vol. 338, 1999, pp. 619- 628.
 D. C. Hornigold, R. Mistry, P. D. Raymond, J. L. Blank, R. A. Challiss, “Evidence for cross-talk between M2 and M3 muscarinic acetylcholine receptors in the regulation of second messenger and extracellular signal-regulated kinase signalling pathways in Chinese hamster ovary cells”. Brithis Journal of Pharmacology, Vol. 138, 2003, pp. 1340-1350.
 D. Zoukhri, E. Macari, S. H. Choi, C. L. Kublin, “c-Jun NH2-terminal kinase mediates interleukin-1beta-induced inhibition of lacrimal gland secretion”. Journal of Neurochemistry, Vol. 96, 2006, pp. 126-135.
 K. Soejima, H. Nakamura, M. Tamai, A. Kawakami, K. Eguchi, “Activation of MKK4 (SEK1), JNK, and c-Jun in labial salivary infiltrating T cells in patients with Sj?gren’s syndrome”. Rheumatology Inernational, Vol. 27, S.