JBM  Vol.5 No.2 , February 2017
Cardiovascular Diseases and Radiations
Abstract: Human beings have continually been exposed to radiation from sources that are terrestrial. There are different sources of radiation for instance ubiquitous background and medical exams that require X-rays. In America for example, the rate of radiation intake per individual increased from 1.6 to 6.2 mSv. The increase has been associated with increased imaging procedures in healthcare facilities. The continued exposure of people to radiant’s increases their rates of developing cardiovascular related diseases. A person who is exposed to low amounts of radiation over a long duration may in the long run develop heart diseases. The result has been obtained from an experiment with the Japanese survivors of the atomic bomb. This research paper focuses on the different sources of radiations and the risk of contracting cardiovascular diseases. The paper also explains the possible relationship between radiations and cardiovascular diseases.
Cite this paper: Sherif, A. , Benhammuda, M. , Fares, S. and Oroszi, T. (2017) Cardiovascular Diseases and Radiations. Journal of Biosciences and Medicines, 5, 72-77. doi: 10.4236/jbm.2017.52007.

[1]   Vrijheid, M., Cardis, E., Ashmore, P., Auvinen, A., Bae, J.M., Engels, H. and Kurtinaitis, J. (2007) Mortality from Diseases Other than Cancer Following Low Doses of Ionizing Radiation: Results from the 15-Country Study of Nuclear Industry Workers. International Journal of Epidemiology, 36, 1126-1135.

[2]   Dale, W.M. (2008) The Effects of Ionizing Radiations on Enzymes in Vitro. Ciba Foundation Symposium—Ionizion Radiations and Cell Metabolism, 25-37.

[3]   Modan, B., Keinan, L., Blumstein, T. and Sadetzki, S. (2000) Cancer Following Cardiac Catheterization in Childhood. International Journal of Epidemiology, 29, 424-428.

[4]   Lotze, M.T. and Tracey, K.J. (2005) High-Mobility Group Box 1 Protein (HMGB1): Nuclear Weapon in the Immune Arsenal. Nature Reviews Immunology, 5, 331-342.

[5]   Spalding, K.L., Arner, E., Westermark, P.O., Bernard, S., Buchholz, B A., Bergmann, O., Blomqvist, L., Hoffstedt, J., Näslund, E., Britton, T., Concha, H., Hassan, M., Rydén, M., Frisén, J. and Arner, P. (2008) Dynamics of Fat Cell Turnover in Humans. Nature, 453, 783-787.

[6]   Henning, R.J., Johnson, G.T., Coyle, J.P. and Harbison, R.D. (2017) Acrolein Can Cause Cardiovascular Disease: A Review. Cardiovascular Toxicology.

[7]   Aronow, W.S. (2014) Association of Peripheral Arterial Disease with All-Cause Mortality, Cardiovascular Mortality and Cardiovascular Events. Angiology: Open Access, 2, e105.

[8]   Rosch, P.J. (2008) Cholesterol Does Not Cause Coronary Heart Disease in Contrast to Stress. Scandinavian Cardiovascular Journal, 42, 244-249.

[9]   Shireman, R.M. (2003) Cholesterol. Role of Cholesterol in Heart Disease. Encyclopedia of Food Sciences and Nutrition, 1243-1250.

[10]   Shimizu, Y., Kodama, K., Nishi, N., Kasagi, F., Suyama, A., Soda, M. and Hayashi, M. (2010) Radiation Exposure and Circulatory Disease Risk: Hiroshima and Nagasaki Atomic Bomb Survivor Data, 1950-2003. BMJ, 340, b5349.

[11]   Arner, P., Bernard, S., Salehpour, M., Possnert, G., Liebl, J., Steier, P., Buchholz, B.A., Eriksson, M., Arner, E., Hauner, H., Skurk, T., Rydén, M., Frayn, K.N. and Spalding, K.L. (2011) Dynamics of Human Adipose Lipid Turnover in Health and Metabolic Disease. Nature, 478, 110-113.